Author: Michael R. Hayden

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Preprints

Length of uninterrupted CAG repeats, independent of polyglutamine size, results in increased somatic instability and hastened age of onset in Huntington disease

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Feeding Schedule And Proteolysis Regulate Autophagic Clearance Of Mutant Huntingtin

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The interaction of aging and oxidative stress contributes to pathogenesis in mouse and human Huntington disease neurons

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Gene expression profiles complement the analysis of genomic modifiers of the clinical onset of Huntington disease

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Axonal ER Ca2+ Release Enhances Miniature, but Reduces Activity-Dependent Glutamate Release in a Huntington Disease Model

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Coupled Control of Distal Axon Integrity and Somal Responses to Axonal Damage by the Palmitoyl Acyltransferase ZDHHC17

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